It is common to experience redness in winter, particularly on the extremities of the body, such as the fingers, ears, and nose, which are delicate areas. This redness is a result of the drop in temperatures felt during the winter months, as well as the accompanying cold and dry wind. This environment is conducive to the disruption of the skin barrier. Studies have indeed shown that the change of season is accompanied by a decrease of about 40% in the levels of ceramides, cholesterol and other lipids in the stratum corneum. This layer can no longer fulfil its protective function properly, resulting in an increase in skin dehydration and water loss.
The fragility of the skin during winter makes it more susceptible to environmental irritants and allergens, which induces a mild skin inflammation. This is characterised by an increase in the levels of interleukin 1-α (IL-α), pro-inflammatory cytokines, as well as cortisol and the number of dermal mast cells. The release of these chemical mediators of inflammation manifests at the macroscopic level as the appearance of skin redness.
These rednesses can also be explained by a change in blood circulation within the body. Indeed, when the ambient temperature decreases, the blood vessels located near the skin's surface contract. This vasoconstriction is a physiological response aimed at reducing body heat loss by decreasing blood flow to the skin. This allows to maintain the body's temperature stable at around 36.6°C (homeothermy) and to prevent hypothermia.
Indeed, during winter, the body is frequently subjected to abrupt changes in temperature due to the significant contrasts between the potentially heavily heated interiors of homes and the outdoors. These environmental changes trigger phenomena of successive vasoconstriction-vasodilation. The latter is characterised by an increase in the diameter of the capillaries and an intensification of blood flow, which visually manifests as the appearance of redness on the face.