The etiological classification of wrinkles is chiefly concerned with their principal cause, rather than with their appearance or behaviour during facial movements.
Wrinkles associated with cutaneous atrophy.
Some wrinkles originate in a progressive atrophy of the skin, characterised by a decrease in the thickness and density of the skin tissues. This phenomenon is linked to a slowdown in cell renewal, reduced fibroblast activity and a loss of extracellular matrix components. The skin then becomes more fragile, less resistant to mechanical stress and more prone to visible lines. These wrinkles are part of the skin’s intrinsic ageing process.
Dynamic and static expression wrinkles.
As mentioned earlier, certain wrinkles are associated with the facial muscle activity. Dynamic expression wrinkles initially emerge from repeated contractions of the facial muscles, whereas static expression lines represent the gradual fixation of these folds over time. This progression reflects the skin’s reduced capacity to deform and subsequently return to its original shape.
Wrinkles associated with skin laxity.
The wrinkles associated with skin laxity are primarily caused by an alteration of the skin’s structural support. The reduction of collagen and elastin fibres, combined with the loss of subcutaneous adipose tissue, results in a progressive sagging of the tissues. The skin is no longer able to effectively counter mechanical forces, which favours the appearance of pronounced folds and furrows.
Sleep-induced wrinkles.
The sleep wrinkles constitute a distinct category associated with repeated mechanical stress exerted on the skin during the night. Prolonged facial pressure against the pillow, night after night, can imprint transient folds which, over time, become permanent. These wrinkles are often vertical, asymmetrical and localised on the cheeks, neck or décolletage.